Thursday, November 21, 2019

Various sources suggest that illness and aging are as a result of loss of mitochondrial function. The loss is due to lack of productivity in the electron chain amidst oxidation. To safeguard the oxidative membrane from being distorted researchers have formulated techniques to issue useful proteins beyond the digestive system through the diet and finally get their way into the cells. Lipid replacement treatment is administered to provide supplements that comprise of adequate amounts of oxidants. Damaged lipids in the cell membranes are combined with undamaged lipids whose concentration is higher than the ones generated by the body.

This procedure assists in rebuilding cellular functions by replacing damaged organelles. Chronic fatigue is caused by deterioration in the mitochondrial function. The new lipid replacement method has been devised to offer patients with undamaged lipids.

Consumed lipids are readily absorbed and transported to tissues

Lipid replacement treatment is possible due to the amounts of lipids found in the various cellular compartments. Orally consumed lipids diffuse to the gut epithelium, merged and transported into the lymph and blood. In no time, the lipid molecules are conveyed from the epithelial cells into the endothelial cells and finally to the lipoproteins carriers and into the red blood cells. Once the lipids are in the cells, lipid transporters distribute certain lipids to cell organelles where certain transport proteins carry them. Undamaged lipids that are conveyed to the mitochondrion are utilized to produce other lipids.

Lipid replacement treatment as an ideal approach

LRT works efficiently and is effective since it can restructure mitochondrial membrane and the gut milieu inclusive of its delicate lining simultaneously. The results of the lipid therapy and the consequent recovery are reliable and profound. Some of the benefits of lipid replacement include:

  • Assist in eliminating damaged tissue
  • Boosts mitochondrial function
  • Reduces fatigue and loss of mitochondrion function
  • Safeguards cellular and mitochondrion membranes against damage

The process assists in improving mitochondrial fitness the same way mitochondrial fitness assists in health management, associated symbiosis, and gut-mediated immune reactivity. Clinical use of LTR typically has the capability of reducing the acute effects of aging on mitochondrion and advance mitochondrion function in different acute diseases, eliminate fatigue, and improve modified states of mucosal immunity through inflammasome-mediated dysbiosis.

Lipid replacement therapy can as well remove damaged lipids through a similar reverse technique that may be conveyed by lipid transfer proteins and enzymes that identify and degrade damaged lipids.

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